Nicotine attenuates arachidonic acid-induced neurotoxicity in cultured spinal cord neurons.

4-Hydroxynonenal induces oxidative stress and death of cultured spinal cord neurons.

Cellular glutathione status modulates polychlorinated biphenyl-induced stress response and apoptosis in vascular endothelial cells.

Cellular glutathione status modulates polychlorinated biphenyl-induced stress response and apoptosis in vascular endothelial cells.

[Anonymous].  2000.  Cellular glutathione status modulates polychlorinated biphenyl-induced stress response and apoptosis in vascular endothelial cells.. Toxicology and applied pharmacology. 166(1):36-42.

Effects of lipids and antioxidants on PCB-mediated dysfunction of vascular endothelial cells (EC).

Fatty acid-mediated activation of vascular endothelial cells.

IL-4 induces apoptosis of endothelial cells through the caspase-3-dependent pathway.

IL-4-induced oxidative stress upregulates VCAM-1 gene expression in human endothelial cells.

Nicotine protects against arachidonic-acid-induced caspase activation, cytochrome c release and apoptosis of cultured spinal cord neurons.

Nicotine protects against arachidonic-acid-induced caspase activation, cytochrome c release and apoptosis of cultured spinal cord neurons.

[Anonymous].  2001.  Nicotine protects against arachidonic-acid-induced caspase activation, cytochrome c release and apoptosis of cultured spinal cord neurons.. Journal of neurochemistry. 76(5):1395-403.

The role of methyl-linoleic acid epoxide and diol metabolites in the amplified toxicity of linoleic acid and polychlorinated biphenyls to vascular endothelial cells.

The role of methyl-linoleic acid epoxide and diol metabolites in the amplified toxicity of linoleic acid and polychlorinated biphenyls to vascular endothelial cells.

[Anonymous].  2001.  The role of methyl-linoleic acid epoxide and diol metabolites in the amplified toxicity of linoleic acid and polychlorinated biphenyls to vascular endothelial cells.. Toxicology and applied pharmacology. 171(3):184-93.

High-energy diets, fatty acids and endothelial cell function: implications for atherosclerosis.